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Original Investigation
March 16, 2023

Efficacy and Safety of Weekly Paclitaxel Plus Vistusertib vs Paclitaxel Alone in Patients With Platinum-Resistant Ovarian High-Grade Serous Carcinoma: The OCTOPUS Multicenter, Phase 2, Randomized Clinical Trial

Author Affiliations
  • 1Gynaecology Unit, The Royal Marsden NHS Foundation Trust, London, United Kingdom
  • 2Division of Clinical Studies, Institute of Cancer Research, London, United Kingdom
  • 3Ovarian Cancer Action Research Centre, Department of Surgery and Cancer, Imperial College London, United Kingdom
  • 4The Christie NHS Foundation Trust and University of Manchester, Manchester, United Kingdom
  • 5Beatson West of Scotland Cancer Centre, Glasgow, United Kingdom
  • 6Sussex Cancer Centre, Royal Sussex County Hospital, Brighton, United Kingdom
  • 7Medical Oncology, Imperial College Healthcare NHS Trust, London, United Kingdom
  • 8Division of Cancer Therapeutics, Institute of Cancer Research, London, United Kingdom
  • 9Department of Histopathology, Imperial College Healthcare NHS Trust, London, United Kingdom
  • 10University Hospital Southampton NHS Foundation Trust, Southampton, United Kingdom
  • 11Research Department of Oncology, UCL Cancer Institute, University College London, London, United Kingdom
  • 12Now with Guy’s and St Thomas’ NHS Foundation Trust, London, United Kingdom
  • 13Cancer Research UK Scotland Centre, University of Edinburgh, Edinburgh, United Kingdom
  • 14CRUK Glasgow Clinical Trials Unit, Institute of Cancer Sciences, University of Glasgow, Glasgow, United Kingdom
JAMA Oncol. 2023;9(5):675-682. doi:10.1001/jamaoncol.2022.7966
Key Points

Question  Does the addition of the dual mTORC inhibitor vistusertib add benefit to weekly paclitaxel in patients with platinum-resistant ovarian high-grade serous carcinoma (PR-HGSC)?

Findings  In this phase 2 randomized clinical trial including 140 patients with PR-HGSC, vistusertib did not increase progression-free survival, overall survival, or response rate when added to weekly paclitaxel.

Meaning  The addition of vistusertib to paclitaxel did not improve clinical outcomes in patients with PR-HGSC.

Abstract

Importance  Patients with platinum-resistant or refractory ovarian high-grade serous carcinoma (PR-HGSC) have a poor prognosis and few therapeutic options. Preclinical studies support targeting PI3K/AKT/mTOR signaling in this setting, and a phase 1 study of the dual mTORC1/mTORC2 inhibitor vistusertib with weekly paclitaxel showed activity.

Objective  To evaluate whether the addition of vistusertib to weekly paclitaxel improves clinical outcomes in patients with PR-HGSC.

Design, Setting, and Participants  This phase 2, double-blind, placebo-controlled multicenter randomized clinical trial recruited patients from UK cancer centers between January 2016 and March 2018. Patients with PR-HGSC of ovarian, fallopian tube, or primary peritoneal origin and with measurable or evaluable disease (Response Evaluation Criteria in Solid Tumors version 1.1 and/or Gynecological Cancer Intergroup cancer antigen 125 criteria) were eligible. There were no restrictions on number of lines of prior therapy. Data analysis was performed from May 2019 to January 2022.

Interventions  Patients were randomized (1:1) to weekly paclitaxel (80 mg/m2 days 1, 8, and 15 of a 28-day cycle) plus oral vistusertib (50 mg twice daily) or placebo.

Main Outcomes and Measures  The primary end point was progression-free survival in the intention-to-treat population. Secondary end points included response rate, overall survival, and quality of life.

Results  A total of 140 patients (median [range] age, 63 [36-86] years; 17.9% with platinum-refractory disease; 53.6% with ≥3 prior therapies) were randomized. In the paclitaxel plus vistusertib vs paclitaxel plus placebo groups, there was no difference in progression-free survival (median, 4.5 vs 4.1 months; hazard ratio [HR], 0.84; 80% CI, 0.67-1.07; 1-sided P = .18), overall survival (median, 9.7 vs 11.1 months; HR, 1.21; 80% CI, 0.91-1.60) or response rate (odds ratio, 0.86; 80% CI, 0.55-1.36). Grade 3 to 4 adverse events were 41.2% (weekly paclitaxel plus vistusertib) vs 36.7% (weekly paclitaxel plus placebo), and there was no difference in quality of life.

Conclusions and Relevance  In this randomized clinical trial of weekly paclitaxel and dual mTORC1/2 inhibition in patients with PR-HGSC, vistusertib did not improve clinical activity of weekly paclitaxel.

Trial Registration  isrctn.org Identifier: ISRCTN16426935

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1 Comment for this article
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Importance of development of antitumor agents for platinum-resistant ovarian cancer in Japan
takuma hayashi, MBBS, DMSci, GMRC, PhD | National Hospital Organization Kyoto Medical Center
A clinical trial conducted by Banerjee et al showed that the addition of mammalian target of rapamycin (mTOR) inhibitor vistusertib to paclitaxel did not improve clinical outcomes in patients with platinum-resistant High-Grade serous cancer (PR-HG-SC).

In Japan, the prescription of PARP inhibitors (olaparib or niraparib) for platinum-sensitive High-Grade Serous carcinoma (HG-SC) or HG-SC with pathogenic variant in BRCA1/2 is covered by health insurance. However, it has been clarified that PARP inhibitors (olaparib or niraparib) are not effective against PR-HG-SC with pathogenic variants in BRCA1/2.

From December 2019 to February 2023, new therapies for a total of 2811 patients
(Ncc oncopanel: 670; F1CDx: 2141) were examined by the cancer gene panel in cancer genomic medicine at a Japanese national university. New therapies for a total of 174 cases of advanced HG-SC, i.e., PR-HG-SC in Japanese patients were tested by cancer genomic panels. As a result of the cancer genome test, the mTOR pathogenic variants were detected in 34 PR-HG-SC cases, the PI3K pathogenic variants were detected in 11 PR-HG-SC cases, the BRCA1/2 pathogenic variants were detected in 23 PR-HG-SC cases, the HRD-related factors pathogenic variants were detected in12 PR-HG-SC cases.

Therefore, PI3K-mTOR signal inhibitors (Everolimus etc.) were orally administered to 34 PR-HG-SC cases with mTOR pathogenic variant and 11 PR-HG-SC cases with PI3K pathogenic variant. Unfortunately, no antitumor effect of Everolimus was observed in all patients (45 cases). Based on the results of this clinical trial in Japan, Japan's Ministry of Health, Labor and Welfare Pharmaceuticals Officers do not recommend or propose the use of PI3K-mTOR signaling inhibitors for PR-HG-SC. The results of the clinical trial conducted in Japan were similar to the results of the clinical trial conducted by Banerjee et al. The development of antitumor agents that are effective against PR-HG-SC is an important issue in the treatment of gynecologic malignancies.

We do not have potential conflicts of interest.

Dr. Takuma Hayashi, Dr. Ikuo Konishi
National Hospital Organization Kyoto Medical Center
CONFLICT OF INTEREST: None Reported
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