Though the eye is afforded a great deal of protection by the bony orbit, contusion injuries of the globe are common. Fists, baseballs, and other low-speed missiles account for most of these nonpenetrating injuries. It has been estimated that missile velocity must exceed 300 feet per second to effect perforation of the human cornea.1 Contusion is produced at slower speeds. There is a vivid description of ocular contusion in Doggart's paper on the adverse effects of boxing.2 Wolff and Zimmerman have recently rekindled interest in the subject by correlating a characteristic deformity of the anterior chamber angle in contused human eyes with the development of a unilateral secondary glaucoma.3 The deformity consists of a longitudinal tear into the face of the ciliary body (Fig 1). Evidently the trabecular meshwork is also damaged, which accounts for the gradual rise in intraocular pressure.
The following excerpt from Wolff and
WEIDENTHAL DT. Experimental Ocular Contusion. Arch Ophthalmol. 1964;71(1):77–81. doi:10.1001/archopht.1964.00970010093015
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