To the Editor.
—In their letter, Parrish and Slomovic1 report two cases relating high axial myopia to intraocular pressure (IOP) elevations after neodymium (Nd)-YAG capsulotomy. An earlier case report by Kurata et al2 supports their observation. It describes a patient with severe myopia and open-angle glaucoma with a pressure rise to 72 mm Hg that was first treated four days after capsulotomy. Only trace aqueous flare was present. The patient had to be hospitalized and suffered progressive visual field loss.The authors'1 speculation of liquid vitreous possibly playing a role in the Nd-YAG IOP rise is shared by others,3 as presented at the Association for Research in Vision and Ophthalmology on May 8, 1985. There is, in fact, experimental evidence that liquid vitreous in physiologic concentrations may impair outflow in vivo and, thus, contribute to the IOP rise after capsulotomy.4 Not implicating "shockwave and debris"
Schubert HD. Capsulotomy, Myopia, and Unexpected Intraocular Pressure Rise. Arch Ophthalmol. 1986;104(1):22–23. doi:10.1001/archopht.1986.01050130026009
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