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December 1988

Retinal Thinning and Regression of Neovascularization

Author Affiliations

Charlottesville, Va

Arch Ophthalmol. 1988;106(12):1646-1647. doi:10.1001/archopht.1988.01060140818003

To the Editor.  —I read with interest the speculations of Dr Glaser1 concerning the mechanism of action of scatter photocoagulation. Dr Glaser suggests that altered cellular interactions within laser scars may be central to involution of neovascularization. The following case is difficult to understand within the framework of Dr Glaser's theoretical constructs.

Report of a Case.  —A 41-year-old woman presented to the Department of Neurology at the University of Virginia, Charlottesville, with a 17-year history of insulin-dependent diabetes mellitus and a four-month history of seizures. Results of physical examination were remarkable only for disc neovascularization in both eyes. A computed tomographic scan demonstrated a left anterior clinoid meningioma. She was referred to the Department of Ophthalmology. Visual acuity was 20/20 OU, and results of examination were normal except for moderate background diabetic retinopathy and neovascularization on the disc (NVD), greater in the left eye than the right (Fig 1).

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