Postintubation tracheal stenosis develops largely as a result of pressure necrosis from the cuff or shaft of an endotracheal or tracheostomy tube. Endotracheal tubes tend to produce cuff lesions at the point of contact in the subglottic trachea. Tracheostomy tubes will produce both cuff lesions and stomal lesions, the latter likely being the result of unsupported ventilator equipment weighing on a tracheostomy. In contrast, high tracheostomy and cricothyroidostomy tubes tend to produce lesions of the subglottic larynx at the level of the cricoid.1,2