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August 1928


Arch Otolaryngol. 1928;8(2):138-143. doi:10.1001/archotol.1928.00620020154002

Two years ago, I discussed lateral sinus thrombosis before the ophthalmologic and otolaryngologic section of the Oklahoma State Medical Association. The paper was presented without manuscript, and a brief was not prepared, so the discussion was not received for publication by the Journal of the Oklahoma State Medical Association. At that time, after several years of observation, I presented conclusions which were, I believe, not generally accepted; namely, (a) all lateral sinus thrombi are aseptic during the formative period; (b) lateral sinus thrombosis does not come as the result of infecting micro-organisms passing directly into the lateral sinus or from an infected embolus entering the sinus by way of the superior petrosal or some other communicating vessel.

I endeavored to show that a thrombus was the result, rather, of extension by contiguity of tissue. Following inflammatory processes within the temporal bone due to infection the membranous sinus becomes

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