Two years ago, I discussed lateral sinus thrombosis before the ophthalmologic and otolaryngologic section of the Oklahoma State Medical Association. The paper was presented without manuscript, and a brief was not prepared, so the discussion was not received for publication by the Journal of the Oklahoma State Medical Association. At that time, after several years of observation, I presented conclusions which were, I believe, not generally accepted; namely, (a) all lateral sinus thrombi are aseptic during the formative period; (b) lateral sinus thrombosis does not come as the result of infecting micro-organisms passing directly into the lateral sinus or from an infected embolus entering the sinus by way of the superior petrosal or some other communicating vessel.
I endeavored to show that a thrombus was the result, rather, of extension by contiguity of tissue. Following inflammatory processes within the temporal bone due to infection the membranous sinus becomes
TODD HC. ASEPTIC CAVERNOUS SINUS THROMBOSIS. Arch Otolaryngol. 1928;8(2):138–143. doi:10.1001/archotol.1928.00620020154002
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