Acute purulent and nonpurulent conditions of the larynx were formerly designated in many different ways. The following terms were used indiscriminately: edema laryngis, edema glottidis, laryngitis submucosa, laryngitis edematosa, laryngitis phlegmonosa, laryngeal abscess, angina laryngea infiltrata, laryngitis seropurulenta and angina laryngea edematosa. Kuttner1 emphasized that the process currently termed laryngeal abscess, as well as a majority of the processes designated by the foregoing terms, arise from similar conditions, and that therefore the majority of these terms are misleading and superfluous. Most contemporary writers agree on this, and the generally accepted opinion now is that abscess formation in the larynx follows a breaking down and pus formation in acute submucous laryngitis. Any irritation such as an acute catarrh, trauma due to a foreign body, or talking too much will first cause an acute submucous laryngitis. Infections following influenza, typhoid fever, scarlet fever, tuberculosis and syphilis can also
KERNAN JD, SCHUGT HP. PRIMARY SUBMUCOUS LARYNGEAL ABSCESSES. Arch Otolaryngol. 1933;17(1):22–29. doi:10.1001/archotol.1933.03570050029003
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