Every form of therapy should preferably, of course, be based on pathogenesis; but when one lacks knowledge of the cause of a disease one has to be content with symptomatic treatment. So far as otosclerosis is concerned, symptomatic treatment has hitherto been the rule, even though recently in cases in which one hoped, ex juvantibus, to be able to trace the cause, some tentative efforts have been made at the alternative. To these trial measures, which so far have gained no greater importance, I shall have reason to return later. A rational effort to remove the symptoms consists in the establishment of an artificial labyrinthine sound fistula.
Anatomic investigations, in conjunction with those of a clinical nature, into the prevalent and severe aural disease called otosclerosis1 have established that the central factor in this syndrome of progressively reduced hearing, often of a hereditary nature, with tinnitus, giddiness and paracusis willisiana,
NASIELL V. MODERN TREATMENT OF OTOSCLEROSIS. Arch Otolaryngol. 1941;33(6):916–925. doi:10.1001/archotol.1941.00660030927002
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