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September 1942


Author Affiliations

From Barnes Hospital and the Department of Otolaryngology, Washington University School of Medicine.

Arch Otolaryngol. 1942;36(3):390-392. doi:10.1001/archotol.1942.03760030086009

REPORT OF CASE  A teamster, aged 41, became hoarse in May 1941. The hoarseness increased and at the end of four months severe cough, stridor and dyspnea were present on exertion, followed later by dysphagia, pain deep in the ears and generalized weakness. There was a loss in weight of 20 pounds (9.1 Kg.) during this period.When first seen at Barnes Hospital in January 1942 (seven and a half months after the onset of the illness), the patient appeared poorly nourished; he was unable to speak above a whisper and was in mild distress, due to inspiratory stridor.The epiglottis was thick, nodular and pale. It was tilted in a position preventing indirect examination of the larynx and encroached on the airway. Direct examination showed the whole interior of the larynx to be white and nodular. The false and true cords were involved to such an extent as to

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