DELAYED or secondary hemorrhage from the healing tonsillar fossa and adenoid site occurs relatively commonly. In the presence of known vascular or hematologic disorders the mechanism is usually apparent. In otherwise healthy persons the occurrence during the healing by secondary intention has been variously reported between 5.0 and 10.0 per cent.1 The hypoprothrombinemic state caused by acetylsalicylic acid administration was carefully studied by Neivert and associates.2 However, the counteractive effects of vitamin K and the supplemental administration of vitamin C have resulted in the reduction but not the elimination of secondary hemorrhage. This was anticipated by Neivert, as such added factors as trauma, menstruation, pregnancy, nutritional deficiency, dyscrasia and infection were considered as contributory causes.
The effects of weather on normal and on abnormal states have been postulated and correlated from the times of Hippocrates. Further studies have been made in relation to many body functions, the most
PEARLMAN SJ, LOEWY A. SECONDARY TONSILLAR AND ADENOID HEMORRHAGE IN RELATION TO BAROMETRIC PRESSURE CHANGES. AMA Arch Otolaryngol. 1951;53(5):527–529. doi:10.1001/archotol.1951.03750050047004
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