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June 1965


Author Affiliations

2122 W 3rd St Los Angeles, Calif

Arch Otolaryngol. 1965;81(6):643. doi:10.1001/archotol.1965.00750050657027

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To the Editor: There is increasing evidence that the perilymph of the inner ear has an elevated protein in labyrinthine otosclerosis. It is possible that this may be due to a stagnation effect of the perilymph in the inner ear. The inner ear is an out-pouching of the cerebrospinal fluid system and is connected to this system only by a narrow channel through which the spinal fluid flows to form perilymph.

It occurred to me that we could obtain further evidence to support or disprove this theory if we could study those cases in which there would seem to be a very widely patent perilymphatic duct. These cases would seem to be in the group which would not be subjected to any stagnation of the perilymph, since the opening between the perilymphatic fluid spaces and the spinal fluid spaces is widely patent.

I would, therefore, like to request that Archives'

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