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February 1969

Otosclerotic Lesion and Cochlear Degeneration

Author Affiliations

Zürich, Switzerland

Arch Otolaryngol. 1969;89(2):364-371. doi:10.1001/archotol.1969.00770020366023

Since INCE the first international symposium on otosclerosis in Detroit in 1961, the histopathology of sensorineural deafness in otosclerosis has been the subject of much discussion at different meetings.1,2

To avoid repetition we will confine our attention to demonstrating and discussing some of the more recent histological findings which are considered to be of significance in the pathogenesis of sensorineural deafness in otosclerosis. These concern mainly the changes in the blood circulation of the inner ear in cases of otosclerosis. The normal blood supply of the membranous otic labyrinth and of the bony otic capsule are well known thanks to the work of Anson and Bast. There exists—and this is important—no anastomosis between the two vascular systems.3-7

Already in the fetus (Fig 1) the endosteum of the bony otic capsule forms a sharp dividing line between the blood supply of the membranous labyrinth and the blood supply

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