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Article
November 1977

Acute Peripheral Facial Palsy: Part of a Cranial Polyneuropathy?

Author Affiliations

From the Departments of Otolaryngology (Dr Djupesland), Bacteriology (Dr Degré), Neurology (Dr Stien), and Clinical Chemistry (Dr Skrede), Rikshospitalet, National Hospital of Norway, University Hospital, Oslo.

Arch Otolaryngol. 1977;103(11):641-644. doi:10.1001/archotol.1977.00780280041004
Abstract

• In 14 of 16 consecutive patients with acute peripheral facial palsy, one or more (up to four) other nerves were involved. The nerves affected in addition to the facial nerve were as follows: trigeminal (ten patients), vestibular (eight), cochlear (six), vagus (one), and upper cervical (five).

Virus was not isolated from any of the patients. A fourfold increase or decrease in complement-fixing antibody titers was present in eight patients (in four, varicella-zoster; in one, varicella-zoster and mumps; in two, cytomegalovirus; in one, mumps). Further, two of the patients with varicella-zoster antibodies showed clinical signs of herpes zoster oticus. About one fourth of all patients had an increase of ESR and of α2-globulins in serum, and two thirds of them had increased γ-globulins in CSF. Acute peripheral facial palsy seems to be part of a cranial polyneuropathy and may be caused by a viral infection.

(Arch Otolaryngol 103:641-644, 1977)

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