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Progress in Pediatrics
October 1929

PATHOGENESIS OF ACUTE SEROFIBRINOUS PLEURISY

Author Affiliations

GOTHENBURG, SWEDEN
From the Medical Department of the Children's Hospital.

Am J Dis Child. 1929;38(4):829-833. doi:10.1001/archpedi.1929.01930100149017
Abstract

Acute serofibrinous pleurisy is now almost invariably regarded as due to tuberculosis. In what manner tuberculosis gives rise to it, is, on the other hand, somewhat obscure. As a rule, one has been content to state that the tuberculous infection in some manner produces the exudation. The most obvious and common explanation is that a subpleural tuberculous focus has provoked an irritation of the pleura.

That a local tuberculous focus in the lung or the bronchial glands constitutes an important factor in the genesis of pleurisy is indicated by the fact that a focus of this kind can be detected on the side on which the exudation subsequently arises. If an opportunity presents itself of examining a child a short time before it contracts pleurisy, there are generally found homolateral hilar changes or a homolateral primary focus. Of fifty children examined before the development of pleurisy, forty-eight showed homolateral changes

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