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A study of the pathology of cerebral lesions in the new-born is based on forty-two autopsies. The author found that cerebral hemorrhage was almost always subarachnoid. Associated with the lesions were vasodilatation, seroalbuminous edema and choroid alteration. He traces the development of repair of these lesions by excellent photomicrographs.
The spinal fluid was studied in three hundred cases. The author found that cerebral hemorrhage was shown by alterations in the spinal fluid, such as hyperalbuminuria, cellular elements and blood cells. Obstetrical factors, such as presentation, premature rupture of the membranes, rigidity of the maternal parts and injections of pituitary extract, were important in the etiology. Syphilis did not play a part in the etiology. From a direct etiologic standpoint, the condition is always a circulatory disorder caused by compression. The clinical condition should be divided into three stages: (1) that of apparent death, (2) coma for twenty-four hours and (3)
Les lésions cérébroméningées à la naissance. Am J Dis Child. 1932;43(1):266–267. doi:10.1001/archpedi.1932.01950010273027
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