MANY infections of childhood have been assigned etiologic responsibility in bronchiectasis. In recent years a few communications have indicated that pathogenic factors often seem to occur during the course of primary tuberculosis and might quite conceivably produce ectasia. In 1933 Jones and Cournand1 discussed the shrunken lobe with bronchiectasis. They carefully described the pathogenic factors involved, mentioning enlarged lymph nodes in primary tuberculosis as being possible factors, particularly in involvement of the middle lobe. Brock and his co-workers2 suggested (1937) that obstructive lesions giving rise to the roentgenologic picture of so-called epituberculosis might eventuate in bronchiectasis. They illustrated this contention with a case history. In 1942 Kent3 provided further evidence by demonstrating residual asymptomatic bronchiectasis in 7 of 10 children who had previously shown roentgenographic evidence of pulmonary atelectasis during the course of primary tuberculosis.
In 1942 a study from the Maybury Sanatorium gave data4 which
JONES EM, PECK WM, WILLIS HS. BRONCHIECTASIS FOLLOWING PRIMARY TUBERCULOSIS. Am J Dis Child. 1946;72(3):296–309. doi:10.1001/archpedi.1946.02020320047006
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