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July 1976

Potter Syndrome

Author Affiliations

Department of Pathology Tripler Army Medical Center APO San Francisco, CA 96438

Am J Dis Child. 1976;130(7):781. doi:10.1001/archpedi.1976.02120080103020

Sir—A recent article by Fantel and Shepard in the Journal (129:1346, 1975) described an infant having stigmata associated with oligohydramnios in the absence of renal dysfunction under the category "Potter syndrome." Potter herself has emphatically written that since renal nonfunction is the basic condition in the syndrome she described, when renal function is present, the condition should not be designated "Potter syndrome."1

The authors propose that chronic leakage of amniotic fluid, in the presence of a normal fetal genitourinary system, caused oligohydramnios and its sequela. This etiology for the oligohydramnios syndrome has been well established in two previous reports,2.3 not cited by Fantel and Shepard.

One of these latter reports3 indicates that vernix granulomatosis of the amnion is "diagnostic for oligohydramnios" of renal or of nonrenal origin. It would, thus, have been instructive had Fantel and Shepard correlated possible placental histopathologic findings with fetal features in

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