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In their study van der Aa et alArticle found that more of the variation in sedentary behavior among 20-year-old individuals was accounted for by genetics (48% for males and 34% for females) and nonshared environmental factors (52% for males and 66% for females) than it was for 12-year-old adolescents (35% for males and 19% for females by genetics and 36% for males and 34% for females by nonshared environmental factors). Conversely, shared environmental factors played less of a role in late adolescence.
Genetic risk of obesity, as identified in single-nucleotide polymorphisms, is unrelated to birth weight, according to a study by Belsky et alArticle. After birth, children at higher genetic risk gained weight more rapidly and reached adiposity rebound earlier and at a greater body mass index than children at lower risk, independent of family history. These phenotypes in turn predicted adult obesity.
This Month in Archives of Pediatrics & Adolescent Medicine. Arch Pediatr Adolesc Med. 2012;166(6):499–500. doi:10.1001/archpediatrics.2011.534
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