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Invited Commentary
May 2016

Transient Acute Kidney Injury in the Postoperative Period: It Is Time to Pay Closer Attention

Author Affiliations
  • 1Division of Vascular Surgery, Harbor-UCLA Medical Center, Torrance, California
JAMA Surg. 2016;151(5):450-451. doi:10.1001/jamasurg.2015.4660

Many of us have had the unsettling experience of watching the serum creatinine level rise or the urine output dwindle following a complex vascular operation. As kidney function deteriorates, we steel ourselves for the inevitable need for dialysis. The second guessing begins. Was it the contrast? The brief intraoperative hypotension? Or, perhaps a nephrotoxic drug? As the creatinine level ebbs, we breathe a sigh of relief. However, a growing body of literature indicates that we are lulled by a false sense of security. As highlighted in the article by Huber et al,1 transient acute kidney injury (AKI) in the postoperative period has lasting, long-term, mortal consequences on the heart.

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